Thromboembolism and Factor VII Defect

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Jaundice and factor VII deficiency in newborn

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Marburg I polymorphism of factor VII-activating protease and risk of venous thromboembolism.

The factor VII–activating protease (FSAP), a newly discovered serine-protease present in human plasma, has 2 main functions in hemostasis: it is a potent activator of prourokinase1 and accelerates coagulation by activating factor VII, independently of tissue factor.2 The FSAP Marburg I polymorphism (1601G A) was recently evaluated as a candidate risk factor for venous thromboembolism (VTE), sin...

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Marburg I polymorphism of factor VII-activating protease and risk of recurrent venous thromboembolism.

Whether a single nucleotide polymorphism (1601 G > A) in the factor VII-activating protease gene (FSAP Marburg I) is a risk factor for venous thromboembolism (VTE) is unclear. We investigated the relevance of the variant with respect to recurrentVTE. 854 patients with a first unprovoked VTE were followed for an average of 41 months after discontinuation of anticoagulation. Study endpoint was sy...

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Preparation of factor VII concentrate using CNBr-activated

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Marburg I polymorphism of factor VII-activating protease is associated with idiopathic venous thromboembolism.

The factor VII-activating protease (FSAP) variant Marburg I is known to attenuate the profibrinolytic system in vitro and was recently shown to be a significant predictor for the evolution and progression of carotid stenosis. The objective of this case-control study was to assess FSAP Marburg I's role in the occurrence of venous thromboembolism (VTE). The frequency of FSAP Marburg I was signifi...

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ژورنال

عنوان ژورنال: Acta Haematologica

سال: 1984

ISSN: 0001-5792,1421-9662

DOI: 10.1159/000206416